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development of IAA was considered very likely as a result of oxidase deamination of NMT (Barker et al., 1980), but was later recognized being also in part by immediate deamination by MAO (Barker et al., 1982). Pretreatment with the MAO-I iproniazid in rat whole Mind homogenates inhibited IAA development by 83%, NMT and DMT-NO development ended up i
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